Manufactured By                                 Latin Name
Dr. JPG Organics                           Phyllanthus emblica

Introduction
The Phyllanthus emblica fruit (also known as Emblica officinalis) or extract from these fruits has been used in traditional medicine for generations to treat symptoms ranging from constipation to the treatment of tumors .
Amla, is arguably the most important medicinal plant in the Indian traditional system of medicine, the Ayurveda. Various parts of the plant are used to treat a range of diseases, but the most important is the fruit. Preclinical studies have shown that amla possesses antipyretic, analgesic, antitussive, antiatherogenic, adaptogenic, cardioprotective, gastroprotective, antianemia, antihypercholesterolemia, wound healing, antidiarrheal, antiatherosclerotic, hepatoprotective, nephroprotective, and neuroprotective properties.

Research

1. The aqueous extract of amla, inhibited the growth of A549 (lung), HepG2 (liver), HeLa (cervical), MDA-MB-231 (breast), SK-OV3 (ovarian), and SW620 (colorectal) cells in vitro.(2)
2. An amla extract possesses antiproliferative activity in MCF7 and MDA-MB-231 breast cancer cell lines and also induces an increase in ERαmRNA in these cells. (3)

Certifications & Notes

• Certified USDA & India Organic
  
• 100% Organic Free From Harmful Chemical, Synthetic Pesticides And Synthetic Fertilizers
• 100% Pure Amla Powder. No Added Colors and Flavors. No Food Additives. No Preservatives.

References
1) https://pubmed.ncbi.nlm.nih.gov/2259214/
2) Ngamkitidechakul C, Jaijoy K, Hansakul P, Soonthornchareonnon N, Sireeratawong S. Antitumour effects of Phyllanthus emblica L: induction of cancer cell apoptosis and inhibition of in vivo tumour promotion and in vitro invasion of human cancer cells Phytother Res. 2010;24:1405–1413

3) Lambertini E, Piva R, Khan MT, Lampronti I, Bianchi N, Borgatti M, Gambari R. Effects of extracts from Bangladeshi medicinal plants on in vitro proliferation of human breast cancer cell lines and expression of estrogen receptor alpha gene Int J Oncol. 2004;24:419–423